October, 2010
Epidemiology vs. Etiology
Over the last several years I’ve been digging into the science of cancer and systems biology, while at the same time looking at the epidemiology of disease and nutrition. And the more I learn, the more I’m convinced that there’s a gap that our scientific tools and methodologies cannot account for. While I’ve discussed this generally under the heading of Science 2.0 (also here), I’ve had a hard time putting into language the exact nature of the gap.
I’ve begun a series of posts that I hope will illustrate the gap, which I believe has to do with the fundamental difference between epidemiology (which is based on statistical observation) and etiology (which seeks to find causal mechanisms for observed phenomena):
- A1 vs. A2 Milk
- Medicine 2.0
- Sunlight vs. Vitamin D
- Animals vs. Plants
- Sugars
Once I’ve completed these posts, I’ll attempt to explain the nature of the gap and what it means for the future of scientific inquiry.…
A1 vs. A2 Milk
Large scale epidemiological studies have linked casein (a cow’s milk protein) to autoimmune disease and heart disease (see The China Study). I just ran across the following twist on this theme which purports to explain a mechanism: a genetic mutation many years ago in domesticated cows transformed the original, safe casein (“A2”) into a toxic form (“A1”). Most cow’s milk available today has both.
What do people think of this? Has anyone tried personally to ingest milk products that are exclusively A2?
Given the dubious connection between cholesterol and heart disease, could the bad rap on cheeses, cream and ice cream have more to do with A1 and sugar than the animal fat and cholesterol?…
Will You Die From Cancer?
Use the arrows that appear if you hover over the bottom right of the Prezi. You may want to expand to full screen first.
All feedback welcome. If you like this, what subject would you like to see addressed next using Prezi?…